George A. Diaz, Robert M. Rakita, David M. We report an immunocompetent patient with recurrent auricular and facial vesicles associated with painful paresthesias and facial paralysis, consistent with Ramsay Hunt syndrome [ 1 ], due to herpes simplex virus HSV type 2. Clinical and laboratory-proven acyclovir resistance developed during therapy. Immunologic assays revealed normal reactivity to HSV
Herpes zoster oticus is herpes zoster virus infection of the clusters of nerve cells ganglia that control the nerves responsible for hearing and balance 8th cranial nerve and for facial movement 7th cranial nerve. Herpes zoster shingles is infection that results from a reactivation of the varicella-zoster virus, the virus that causes chickenpox. After an episode of chickenpox, this virus lies dormant in nerve roots and can be reactivated, travelling down the nerve fibers to the skin, where it causes painful sores. Most often the cause of reactivation is unknown but sometimes it occurs when the immune system is weakened, for example, by cancer, AIDS, or certain drugs. Herpes zoster oticus occurs when the herpes zoster virus is reactivated in the 7th facial and 8th cranial nerves.
Facial herpes is very common and is also known as cold sores, fever blisters, sun blisters, oro-facial herpes, herpes labialis and herpes febrilis. Facial herpes is characterised by groups of fluid-filled blisters that appear on red swollen areas of the skin or on the mucous membranes. A burning sensation is often present just before the skin lesions develop. The areas can be tender and painful.
Piper, M. Petri, M. Herpes zoster is a common nervous disease known since ancient times. The clinical manifestations are a vesicular cutaneous eruption, radicular neuralgia and, less often segmental palsies and sensory loss